#praxia

• Dexterous / Skilful — The quality of an individual to demonstrate motor dexterity, fine coordination, and speed in executing complex physical or cognitive tasks. In neurological semiology and occupational medicine, the term evaluates the integrity of the pyramidal and extrapyramidal tracts; an 'able' or 'skilful' patient presents intact reflexes, preserved praxia (the ability to execute coordinated intentional movements), and the absence of intentional tremor or ataxia.
• Apractognosia — A complex neuropsychological syndrome characterized by the coexistence of apraxia (the inability to execute coordinated and intentional movements) and agnosia (the inability to recognize sensory stimuli, objects, or spatial patterns), despite intact elementary motor and sensory functions. This structural phenomenon is usually caused by focal lesions located in the non-dominant (right) parietal lobe of the cerebral cortex, severely affecting spatial orientation and body schemas.
• Apractophagie — A severe neuropsychological disorder, considered a specific form of ideomotor apraxia, manifested by the loss of the ability to execute the coordinated motor sequences required for eating (such as chewing, using utensils, or bringing food to the mouth), despite intact elementary swallowing reflexes and muscle functions. This structural phenomenon is usually associated with extensive lesions of the frontal or parietal lobe.
• Apraxic — Suffering from apraxia or pertaining to apraxia; a person affected by the loss of the ability to execute voluntary coordinated and goal-directed movements, despite intact basic motor and sensory functions. In a structural clinical sense, the term describes clinical manifestations, behaviors, or cerebral lesions associated with the inability to plan and sequence movement schemas.
• Apraxia — A neurological disorder characterized by an individual's pathological inability to execute coordinated, intentional, and goal-directed movements or gestures, despite completely intact elementary motor functions (strength, tone, reflexes) and sensory pathways. This structural phenomenon is caused by lesions located in the association areas of the cerebral cortex, particularly in the parietal or frontal lobe.